[Molecular analyses of human influenza viruses. Circulation of new variants since 1995/96].
Identifieur interne : 000432 ( Main/Exploration ); précédent : 000431; suivant : 000433[Molecular analyses of human influenza viruses. Circulation of new variants since 1995/96].
Auteurs : B. Biere [Allemagne] ; B. SchweigerSource :
- Bundesgesundheitsblatt, Gesundheitsforschung, Gesundheitsschutz [ 1436-9990 ] ; 2008.
Descripteurs français
- KwdFr :
- Allemagne (MeSH), Grippe humaine (immunologie), Grippe humaine (prévention et contrôle), Grippe humaine (virologie), Gènes viraux (génétique), Humains (MeSH), Hémagglutinines virales (génétique), Hémagglutinines virales (immunologie), Phylogenèse (MeSH), Sialidase (génétique), Sialidase (immunologie), Vaccins antigrippaux (immunologie), Virus de la grippe A (génétique), Virus de la grippe A (immunologie), Virus influenza B (génétique), Virus influenza B (immunologie), Virus recombinants (génétique), Virus recombinants (immunologie), Épitopes (génétique), Épitopes (immunologie).
- MESH :
- génétique : Gènes viraux, Hémagglutinines virales, Sialidase, Virus de la grippe A, Virus influenza B, Virus recombinants, Épitopes.
- immunologie : Grippe humaine, Hémagglutinines virales, Sialidase, Vaccins antigrippaux, Virus de la grippe A, Virus influenza B, Virus recombinants, Épitopes.
- prévention et contrôle : Grippe humaine.
- virologie : Grippe humaine.
- Allemagne, Humains, Phylogenèse.
- Wicri :
- geographic : Allemagne.
English descriptors
- KwdEn :
- Epitopes (genetics), Epitopes (immunology), Genes, Viral (genetics), Germany (MeSH), Hemagglutinins, Viral (genetics), Hemagglutinins, Viral (immunology), Humans (MeSH), Influenza A virus (genetics), Influenza A virus (immunology), Influenza B virus (genetics), Influenza B virus (immunology), Influenza Vaccines (immunology), Influenza, Human (immunology), Influenza, Human (prevention & control), Influenza, Human (virology), Neuraminidase (genetics), Neuraminidase (immunology), Phylogeny (MeSH), Reassortant Viruses (genetics), Reassortant Viruses (immunology).
- MESH :
- chemical , genetics : Epitopes, Hemagglutinins, Viral, Neuraminidase.
- chemical , immunology : Epitopes, Hemagglutinins, Viral, Influenza Vaccines, Neuraminidase.
- geographic : Germany.
- genetics : Genes, Viral, Influenza A virus, Influenza B virus, Reassortant Viruses.
- immunology : Influenza A virus, Influenza B virus, Influenza, Human, Reassortant Viruses.
- prevention & control : Influenza, Human.
- virology : Influenza, Human.
- Humans, Phylogeny.
Abstract
The evolution of influenza viruses is increasingly pursued by molecular analyses that complement classical methods. The analyses focus on the two surface proteins hemagglutinin (HA) and neuraminidase (NA) which determine the viral antigenic profile. Influenza A(H3N2) viruses are exceptionally variable, so that usually at least two virus variants cocirculate at the same time. Together with influenza B viruses they caused approximately 90% of influenza virus infections in Germany during the last 12 seasons, while influenza A(H1N1) viruses only played a subordinate part. Unexpectedly, reassorted viruses of subtype A(H1N2) appeared during the seasons 2001/02 and 2002/03, but were isolated only rarely and gained no epidemiological significance. Furthermore, during the season 2001/02 influenza B viruses of the Victoria-lineage reappeared in Germany and other countries of the northern hemisphere after 10 years of absence. These viruses reassorted with the cocirculating Yamagata-like influenza B viruses, as could be seen by the appearance of viruses with a Victoria-like HA and a Yamagata-like NA.
DOI: 10.1007/s00103-008-0634-2
PubMed: 18773168
Affiliations:
Links toward previous steps (curation, corpus...)
Le document en format XML
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Circulation of new variants since 1995/96].</ArticleTitle><Pagination><MedlinePgn>1050-60</MedlinePgn></Pagination><ELocationID EIdType="doi" ValidYN="Y">10.1007/s00103-008-0634-2</ELocationID><Abstract><AbstractText>The evolution of influenza viruses is increasingly pursued by molecular analyses that complement classical methods. The analyses focus on the two surface proteins hemagglutinin (HA) and neuraminidase (NA) which determine the viral antigenic profile. Influenza A(H3N2) viruses are exceptionally variable, so that usually at least two virus variants cocirculate at the same time. Together with influenza B viruses they caused approximately 90% of influenza virus infections in Germany during the last 12 seasons, while influenza A(H1N1) viruses only played a subordinate part. Unexpectedly, reassorted viruses of subtype A(H1N2) appeared during the seasons 2001/02 and 2002/03, but were isolated only rarely and gained no epidemiological significance. Furthermore, during the season 2001/02 influenza B viruses of the Victoria-lineage reappeared in Germany and other countries of the northern hemisphere after 10 years of absence. These viruses reassorted with the cocirculating Yamagata-like influenza B viruses, as could be seen by the appearance of viruses with a Victoria-like HA and a Yamagata-like NA.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Biere</LastName><ForeName>B</ForeName><Initials>B</Initials><AffiliationInfo><Affiliation>Robert Koch-Institut, Berlin, BRD.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Schweiger</LastName><ForeName>B</ForeName><Initials>B</Initials></Author></AuthorList><Language>ger</Language><PublicationTypeList><PublicationType UI="D004740">English Abstract</PublicationType><PublicationType UI="D016428">Journal Article</PublicationType></PublicationTypeList><VernacularTitle>Molekulare Analyse humaner Influenzaviren. 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Schweiger</name></noCountry><country name="Allemagne"><region name="Berlin"><name sortKey="Biere, B" sort="Biere, B" uniqKey="Biere B" first="B" last="Biere">B. Biere</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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